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darüber gibts auch schon was bei uns im Forum, zB: hier.... Das produzierte Gift heißt Nagalase, GcMAF ist der Schlüssel zur Aktivierung des Immunsystems.
Den Rest müsst ihr euch ergoogeln.![]()
Ich auch nicht, darleen.
Da ich aber auch andere Medikamente nicht vertrage, nehme ich an, daß es da einen gemeinsamen Nenner gibt. Den zu finden, ist die eine Sache. Ihn zu beheben, die andere.
Grüsse,
Oregano
Zum Marshal Protokoll gibt es im Forum viele Infos. - Bitte nachlesen.
Der Gedanke, über einen längeren Zeitpunkt ABs einzunehmen, ist für mich gar nicht verlockend.
Gruss,
Oregano
Vitamin D toxicity redefined: vitamin K and t... [Med Hypotheses. 2007] - PubMed - NCBIVitamin D toxicity redefined: vitamin K and the molecular mechanism.
Masterjohn C.
...
Abstract
The dose of vitamin D that some researchers recommend as optimally therapeutic exceeds that officially recognized as safe by a factor of two; it is therefore important to determine the precise mechanism by which excessive doses of vitamin D exert toxicity so that physicians and other health care practitioners may understand how to use optimally therapeutic doses of this vitamin without the risk of adverse effects. Although the toxicity of vitamin D has conventionally been attributed to its induction of hypercalcemia, animal studies show that the toxic endpoints observed in response to hypervitaminosis D such as anorexia, lethargy, growth retardation, bone resorption, soft tissue calcification, and death can be dissociated from the hypercalcemia that usually accompanies them, demanding that an alternative explanation for the mechanism of vitamin D toxicity be developed. The hypothesis presented in this paper proposes the novel understanding that vitamin D exerts toxicity by inducing a deficiency of vitamin K. According to this model, vitamin D increases the expression of proteins whose activation depends on vitamin K-mediated carboxylation; as the demand for carboxylation increases, the pool of vitamin K is depleted. Since vitamin K is essential to the nervous system and plays important roles in protecting against bone loss and calcification of the peripheral soft tissues, its deficiency results in the symptoms associated with hypervitaminosis D. This hypothesis is circumstantially supported by the observation that animals deficient in vitamin K or vitamin K-dependent proteins exhibit remarkable similarities to animals fed toxic doses of vitamin D, and the observation that vitamin D and the vitamin K-inhibitor Warfarin have similar toxicity profiles and exert toxicity synergistically when combined. The hypothesis further proposes that vitamin A protects against the toxicity of vitamin D by decreasing the expression of vitamin K-dependent proteins and thereby exerting a vitamin K-sparing effect. If animal experiments can confirm this hypothesis, the models by which the maximum safe dose is determined would need to be revised. Physicians and other health care practitioners would be able to treat patients with doses of vitamin D that possess greater therapeutic value than those currently being used while avoiding the risk of adverse effects by administering vitamin D together with vitamins A and K.
Cenaverde B.V. Nahrungsergänzungsmittel: Starke und gesunde Knochen mit MenaQ-7 Vitamin K2 und D3MenaQ-7: Vitamine K2 und D3
Neuste Erkenntnisse zeigen: Vitamin K2 ist notwendig, um die Steuerungs-Proteine für den Calciumstoffwechsel zu aktivieren. Bei ausreichender Versorgung wird das Calcium nicht in den Arterien abgelagert, sondern gelangt in die Knochen. MenaQ-7 enthält eine hohe Menge von natürlichem Vitamin K2. Es wird aus dem Extrakt der fermentierten Sojabohne (Natto) gewonnen.
Daß das Produkt aus der Sojabohne gewonnen wird, ist mir nicht sonderlich sympathisch. Da halte ich mich doch zunächst lieber an die Lebensmittel, die reichlich Vitamin K enthalten.
Warum ich es so schlecht vertrage weiß ich nicht, nehme auch täglich Magnesium.
Übrigens habe ich und mein Bekannter Borreliose.